Muscle-derived myostatin is a major endocrine driver of follicle-stimulating hormone synthesis.

Document Type

Article

Publication Date

1-17-2025

Publication Title

Science

Keywords

Animals, Myostatin, Mice, Female, Follicle Stimulating Hormone, Muscle, Skeletal, Pituitary Gland, Activins, Ovary, Fertility, Mice, Inbred C57BL

JAX Source

Science. 2025;387(6731):329-36.

Volume

387

Issue

6731

First Page

329

Last Page

336

ISSN

1095-9203

PMID

39818879

DOI

https://doi.org/10.1126/science.adi4736

Abstract

Myostatin is a paracrine myokine that regulates muscle mass in a variety of species, including humans. In this work, we report a functional role for myostatin as an endocrine hormone that directly promotes pituitary follicle-stimulating hormone (FSH) synthesis and thereby ovarian function in mice. Previously, this FSH-stimulating role was attributed to other members of the transforming growth factor-β family, the activins. Our results both challenge activin's eponymous role in FSH synthesis and establish an unexpected endocrine axis between skeletal muscle and the pituitary gland. Our data also suggest that efforts to antagonize myostatin to increase muscle mass may have unintended consequences on fertility.

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