Faculty Research 1990 - 1999

Neuromuscular degeneration (nmd): a mutation on mouse chromosome 19 that causes motor neuron degeneration.

Document Type

Article

Publication Date

1995

Keywords

Chromosome-Mapping: mt, Crosses-Genetic, Female, Glycogen-Phosphorylase: ge, Male, Mice, Mice-Inbred-CBA, Mice-Inbred-C57BL, Motor-Neurons: ph, pa, Muscle-Skeletal: pa, Muscular-Atrophy-Spinal: ge, pa, Mutation: ge, Nerve-Degeneration: ge, Nerve-Growth-Factors: ge, Nerve-Tissue-Proteins: ge, Spinal-Cord: pa, SUPPORT-U-S-GOVT-P-H-S

First Page

187

Last Page

191

JAX Source

Mamm Genome 1995 Mar;6(3):187-91

Grant

RR01183/RR/NCRR, GM46697/GM/NIGMS, CA34196/CA/NCI

Abstract

Neuromuscular degeneration, nmd, is a spontaneous autosomal recessive mutation in the mouse producing progressive hindlimb impairment caused by spinal muscular atrophy. We used an intersubspecific intercross between B6.BKs-nmd2J/+ and Mus musculus castaneus (CAST/Ei) to map the nmd mutation to mouse Chromosome (Chr) 19 with the most likely gene order: nmd-(D19Sel2, Pygm)-Cntf-Pomc2-D19Mit16-Cyp2c-Got1. nmd maps near muscle deficient, mdf, and has a very similar clinical phenotype, but allele tests and histological differences suggest that nmd is a distinct mutation at a different locus. Although closely linked, nmd recombined with the candidate genes muscle glycogen phosphorylase, Pygm, and ciliary neurotrophic factor, Cntf.

Please contact the Joan Staats Library for information regarding this document.

Share

COinS