The Th1/Tfh-like biased responses elicited by the rASP-1 innate adjuvant are dependent on TRIF and Type I IFN receptor pathways.
JGM, Adaptor Proteins, Vesicular Transport, Adjuvants, Immunologic, Adjuvants, Pharmaceutic, Animals, Chemokine CXCL10, Humans, Influenza A Virus, H1N1 Subtype, Influenza Vaccines, Interleukin-12 Subunit p40, Interleukin-17, Mice, Myeloid Differentiation Factor 88, Receptor, Interferon alpha-beta, Toll-Like Receptor 4, Tumor Necrosis Factor-alpha
Front Immunol. 2022;13:961094.
This research was funded by NIH grants U01AI124260 and U01AI160421.
The Th1/Tfh-like biased responses elicited by the rASP-1 innate adjuvant are dependent on TRIF and Type I IFN receptor pathways. Front Immunol. 2022;13:961094.
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