Disease Progression in Children with Perinatal HIV Correlates with Increased PD-1+ CD8 T Cells that Coexpress Multiple Immune Checkpoints.
Document Type
Article
Publication Date
11-15-2021
Publication Title
The Journal of infectious diseases
Keywords
JGM
JAX Source
J Infect Dis 2021 Nov 15; 224:1785-95
Volume
224
First Page
1785
Last Page
1795
ISSN
1537-6613
PMID
33864071
DOI
https://doi.org/10.1093/infdis/jiab204
Abstract
BACKGROUND: PD-1 marks exhausted T cells, with weak effector functions. Adults living with HIV have increased levels of PD-1+ CD8 T cells that correlate with HIV disease progression, yet little is known about the role of PD-1+ CD8 T cells in children with perinatal HIV.
METHODS: We enrolled 76 Kenyan children with perinatal HIV and 43 children who were HIV unexposed and quantified PD-1 levels on CD8 T cells, their coexpression with immune checkpoints (IC) 2B4, CD160 and TIM3, correlates with immune activation and HIV disease progression and HIV-specific and non-specific proliferative responses.
RESULTS: PD-1+ CD8 T cell frequencies are elevated in children with perinatal HIV and associated with disease progression. The majority of PD-1+ CD8 T cells coexpress additional ICs. ART initiation lowers total PD-1 levels and coexpression of multiple ICs. The frequency of PD-1 + 2B4+CD160+TIM3- in PD-1+ CD8 T cells, predicts weaker HIV-specific proliferative responses, suggesting this subset is functionally exhausted.
CONCLUSION: Children with perinatal HIV have high PD-1+ CD8 T cells that are a heterogeneous population differentially coexpressing multiple ICs. Understanding the complex interplay of ICs is essential to guide the development of PD-1 directed immunotherapies for pediatric HIV remission and cure.
Recommended Citation
Tailor J,
Foldi J,
Generoso M,
McCarty B,
Alankar A,
Kilberg M,
Mwamzuka M,
Marshed F,
Ahmed A,
Liu M,
Borkowsky W,
Unutmaz D,
Khaitan A.
Disease Progression in Children with Perinatal HIV Correlates with Increased PD-1+ CD8 T Cells that Coexpress Multiple Immune Checkpoints. J Infect Dis 2021 Nov 15; 224:1785-95