Document Type

Article

Publication Date

12-1-2023

Keywords

JGM, Humans, Ataxia Telangiectasia, Ataxia Telangiectasia Mutated Proteins, Gene Editing, Induced Pluripotent Stem Cells, Mutation, Cell Cycle Proteins

JAX Source

Stem Cell Res. 2023;73:103247

ISSN

1876-7753

PMID

37976651

DOI

https://doi.org/10.1016/j.scr.2023.103247

Grant

This research received financial support from the Deutsche Forschungsgemeinschaft, grant number IV 21/17-1.

Abstract

Ataxia-Telangiectasia (A-T) is an autosomal recessive multi-system disorder caused by mutations in the ataxia-telangiectasia mutated (ATM) gene, resulting, among other symptoms, in neurological dysfunction. ATM is known to be a master controller of signal transduction for DNA damage response, with additional functions that are poorly understood. CRISPR/Cas9 technology was used to introduce biallelic mutations at selected sites of the ATM gene in human induced pluripotent stem cells (hiPSCs). This panel of hiPSCs with nonsense and missense mutations in ATM can help understand the molecular basis of A-T.

Comments

This is an open access article under the CC BY-NC-ND license (http://creativecommons.org/licenses/by- nc-nd/4.0/).

Share

COinS