Document Type

Article

Publication Date

10-24-2024

Keywords

JGM, JMG, Animals, COVID-19, Disease Models, Animal, Mice, SARS-CoV-2, Lung, Collaborative Cross Mice, Viral Load, Female, Cytokines, Humans, Male

JAX Source

Sci Rep. 2024;14(1):25147.

ISSN

2045-2322

PMID

39448712

DOI

https://doi.org/10.1038/s41598-024-77087-1

Grant

Funding was provided by a grant to the Special Mouse Strain Resources at The Jackson Laboratory from the Na- tional Institutes of Health (NIH), Office of Research Infrastructure Programs (P40 OD011102) and by support from the F. M. Kirby Foundation and Anonymous Family Fund to the Trudeau Institute.

Abstract

Mutations in SARS-CoV-2 variants of concern (VOCs) have expanded the viral host range beyond primates, and a few other mammals, to mice, affording the opportunity to exploit genetically diverse mouse panels to model the broad spectrum of responses to infection in patient populations. Here we surveyed responses to VOC infection in genetically diverse Collaborative Cross (CC) founder strains. Infection of wild-derived CC founder strains produced a broad range of viral burden, disease susceptibility and survival, whereas most other strains were resistant to disease despite measurable lung viral titers. In particular, CAST/EiJ, a wild-derived strain, developed high lung viral burdens, more severe lung pathology than seen in other CC strains, and a dysregulated cytokine profile resulting in morbidity and mortality. These inbred mouse strains may serve as a valuable platform to evaluate therapeutic countermeasures against severe COVID-19 and other coronavirus pandemics in the future.

Creative Commons License

Creative Commons Attribution 4.0 International License
This work is licensed under a Creative Commons Attribution 4.0 International License.

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